EGFR mutation-induced alternative splicing of Max contributes to growth of glycolytic tumors in brain cancer

Alternative splicing contributes to diverse aspects of cancer pathogenesis including altered cellular metabolism, but the specificity of the process or its consequences are not well understood. We characterized genome-wide alternative splicing induced by the activating EGFRvIII mutation in glioblast...

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書誌詳細
主要な著者: Babic, Ivan, Anderson, Erik S., Tanaka, Kazuhiro, Guo, Deliang, Masui, Kenta, Li, Bing, Zhu, Shaojun, Gu, Yuchao, Villa, Genaro, Akhavan, David, Nathanson, David, Gini, Beatrice, Mareninov, Sergey, Li, Rui, Espindola C., Carolina, Kurdistani, Siavash K., Eskin, Ascia, Nelson, Stanley F., Yong, William H., Cavenee, Webster K., Cloughesy, Timothy F., Christofk, Heather R., Black, Douglas L., Mischel, Paul S.
フォーマット: Artigo
言語:Inglês
出版事項: 2013
主題:
Article
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC3679227/
https://ncbi.nlm.nih.gov/pubmed/23707073
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cmet.2013.04.013
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