Oligodendrocyte-Specific Activation of PERK Signaling Protects Mice against Experimental Autoimmune Encephalomyelitis

There is compelling evidence that oligodendrocyte apoptosis, in response to CNS inflammation, contributes significantly to the development of the demyelinating disorder multiple sclerosis and its animal model, experimental autoimmune encephalomyelitis (EAE). Therefore, approaches designed to protect...

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Главные авторы: Lin, Wensheng, Lin, Yifeng, Li, Jin, Fenstermaker, Ali G., Way, Sharon W., Clayton, Benjamin, Jamison, Stephanie, Harding, Heather P., Ron, David, Popko, Brian
Формат: Artigo
Язык:Inglês
Опубликовано: Society for Neuroscience 2013
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Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC3654380/
https://ncbi.nlm.nih.gov/pubmed/23554479
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.1636-12.2013
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