Oligodendrocyte-Specific Activation of PERK Signaling Protects Mice against Experimental Autoimmune Encephalomyelitis

There is compelling evidence that oligodendrocyte apoptosis, in response to CNS inflammation, contributes significantly to the development of the demyelinating disorder multiple sclerosis and its animal model, experimental autoimmune encephalomyelitis (EAE). Therefore, approaches designed to protect...

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Detalhes bibliográficos
Main Authors: Lin, Wensheng, Lin, Yifeng, Li, Jin, Fenstermaker, Ali G., Way, Sharon W., Clayton, Benjamin, Jamison, Stephanie, Harding, Heather P., Ron, David, Popko, Brian
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 2013
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Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3654380/
https://ncbi.nlm.nih.gov/pubmed/23554479
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.1636-12.2013
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