Oligodendrocyte-Specific Activation of PERK Signaling Protects Mice against Experimental Autoimmune Encephalomyelitis

There is compelling evidence that oligodendrocyte apoptosis, in response to CNS inflammation, contributes significantly to the development of the demyelinating disorder multiple sclerosis and its animal model, experimental autoimmune encephalomyelitis (EAE). Therefore, approaches designed to protect...

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Bibliografiske detaljer
Main Authors: Lin, Wensheng, Lin, Yifeng, Li, Jin, Fenstermaker, Ali G., Way, Sharon W., Clayton, Benjamin, Jamison, Stephanie, Harding, Heather P., Ron, David, Popko, Brian
Format: Artigo
Sprog:Inglês
Udgivet: Society for Neuroscience 2013
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Articles
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3654380/
https://ncbi.nlm.nih.gov/pubmed/23554479
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.1636-12.2013
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