Oligodendrocyte-Specific Activation of PERK Signaling Protects Mice against Experimental Autoimmune Encephalomyelitis

There is compelling evidence that oligodendrocyte apoptosis, in response to CNS inflammation, contributes significantly to the development of the demyelinating disorder multiple sclerosis and its animal model, experimental autoimmune encephalomyelitis (EAE). Therefore, approaches designed to protect...

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書誌詳細
主要な著者: Lin, Wensheng, Lin, Yifeng, Li, Jin, Fenstermaker, Ali G., Way, Sharon W., Clayton, Benjamin, Jamison, Stephanie, Harding, Heather P., Ron, David, Popko, Brian
フォーマット: Artigo
言語:Inglês
出版事項: Society for Neuroscience 2013
主題:
Articles
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC3654380/
https://ncbi.nlm.nih.gov/pubmed/23554479
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.1636-12.2013
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