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The Carboxyl Tail of Alpha-Actinin-4 Regulates Its Susceptibility to m-Calpain and thus Functions in Cell Migration and Spreading
Alpha-actinin-4 links the cytoskeleton to sites of adhesion and has been shown to be modulated to enable cell migration. Such focal adhesions must be labile to accomplish migration, with this detachment occurring at least in part via m-calpain activation (Glading et al., 2002, Glading et al., 2001,...
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| Autori principali: | , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2013
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3633689/ https://ncbi.nlm.nih.gov/pubmed/23466492 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.biocel.2013.02.015 |
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