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SH1 domain autophosphorylation of P210 BCR/ABL is required for transformation but not growth factor independence.

P210 BCR/ABL is a chimeric oncogene implicated in the pathogenesis of chronic myelogenous leukemia. BCR sequences have been shown to be required for activation of the tyrosine kinase and transforming functions of BCR/ABL. In this work, we show that two other structural requirements for full transfor...

Täydet tiedot

Tallennettuna:
Julkaisussa:Mol Cell Biol
Päätekijät: Pendergast, A M, Gishizky, M L, Havlik, M H, Witte, O N
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Taylor & Francis 1993
Aiheet:
Research Article
Linkit:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC359485/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8441409/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.13.3.1728-1736.1993
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