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c-Jun N-terminal kinase activation contributes to reduced connexin43 and development of atrial arrhythmias
AIMS: c-Jun N-terminal kinase (JNK) activation is implicated in cardiovascular diseases and ageing, which are linked to enhanced propensity to atrial fibrillation (AF). However, the contribution of JNK to AF remains unknown. Thus, we assessed the role of JNK in remodelling of gap junction connexin43...
Tallennettuna:
| Päätekijät: | , , , , , , |
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| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
Oxford University Press
2013
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3567788/ https://ncbi.nlm.nih.gov/pubmed/23241357 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/cvr/cvs366 |
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