Presynaptic Ca(V)2.1 calcium channels carrying familial hemiplegic migraine mutation R192Q allow faster recovery from synaptic depression in mouse calyx of Held

Samankaltaisia teoksia

• Functional Compensation of P/Q by N-Type Channels Blocks Short-Term Plasticity at the Calyx of Held Presynaptic Terminal
  Tekijä: Inchauspe, Carlota González, et al.
  Julkaistu: (2004)
• Acid-Sensing Ion Channels Activated by Evoked Released Protons Modulate Synaptic Transmission at the Mouse Calyx of Held Synapse
  Tekijä: González-Inchauspe, Carlota, et al.
  Julkaistu: (2017)
• Changes in synaptic transmission properties due to the expression of N-type calcium channels at the calyx of Held synapse of mice lacking P/Q-type calcium channels
  Tekijä: Inchauspe, Carlota González, et al.
  Julkaistu: (2007)
• Calcium channels and synaptic transmission in familial hemiplegic migraine type 1 animal models
  Tekijä: Uchitel, Osvaldo D., et al.
  Julkaistu: (2013)
• Synaptic Gain-of-Function Effects of Mutant Ca(v)2.1 Channels in a Mouse Model of Familial Hemiplegic Migraine Are Due to Increased Basal [Ca(2+)](i)
  Tekijä: Di Guilmi, Mariano N., et al.
  Julkaistu: (2014)