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Is the mitochondrial outermembrane protein VDAC1 therapeutic target for Alzheimer’s disease?
Mitochondrial dysfunction and synaptic damage have been described as early events in Alzheimer’s disease (AD) pathogenesis. Recent research using AD postmortem brains, and AD mouse and cell models revealed that amyloid beta (Aβ) and tau hyperphosphorylation are involved in mitochondrial dysfunction...
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| Autor principal: | |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2012
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3518645/ https://ncbi.nlm.nih.gov/pubmed/22995655 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbadis.2012.09.003 |
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