Genetic KCa3.1-Deficiency Produces Locomotor Hyperactivity and Alterations in Cerebral Monoamine Levels

BACKGROUND: The calmodulin/calcium-activated K(+) channel KCa3.1 is expressed in red and white blood cells, epithelia and endothelia, and possibly central and peripheral neurons. However, our knowledge about its contribution to neurological functions and behavior is incomplete. Here, we investigated...

Disgrifiad llawn

Wedi'i Gadw mewn:
Manylion Llyfryddiaeth
Prif Awduron: Lambertsen, Kate Lykke, Gramsbergen, Jan Bert, Sivasaravanaparan, Mithula, Ditzel, Nicholas, Sevelsted-Møller, Linda Maria, Oliván-Viguera, Aida, Rabjerg, Maj, Wulff, Heike, Köhler, Ralf
Fformat: Artigo
Iaith:Inglês
Cyhoeddwyd: Public Library of Science 2012
Pynciau:
Research Article
Mynediad Ar-lein:https://ncbi.nlm.nih.gov/pmc/articles/PMC3471871/
https://ncbi.nlm.nih.gov/pubmed/23077667
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0047744
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