Genetic KCa3.1-Deficiency Produces Locomotor Hyperactivity and Alterations in Cerebral Monoamine Levels

BACKGROUND: The calmodulin/calcium-activated K(+) channel KCa3.1 is expressed in red and white blood cells, epithelia and endothelia, and possibly central and peripheral neurons. However, our knowledge about its contribution to neurological functions and behavior is incomplete. Here, we investigated...

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Autori principali: Lambertsen, Kate Lykke, Gramsbergen, Jan Bert, Sivasaravanaparan, Mithula, Ditzel, Nicholas, Sevelsted-Møller, Linda Maria, Oliván-Viguera, Aida, Rabjerg, Maj, Wulff, Heike, Köhler, Ralf
Natura: Artigo
Lingua:Inglês
Pubblicazione: Public Library of Science 2012
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Research Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC3471871/
https://ncbi.nlm.nih.gov/pubmed/23077667
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0047744
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