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Repair of cisplatin-induced DNA interstrand crosslinks by a replication-independent pathway involving transcription-coupled repair and translesion synthesis

DNA interstrand crosslinks (ICLs) formed by antitumor agents, such as cisplatin or mitomycin C, are highly cytotoxic DNA lesions. Their repair is believed to be triggered primarily by the stalling of replication forks at ICLs in S-phase. There is, however, increasing evidence that ICL repair can als...

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Bibliografski detalji
Glavni autori: Enoiu, Milica, Jiricny, Josef, Schärer, Orlando D.
Format: Artigo
Jezik:Inglês
Izdano: Oxford University Press 2012
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3467066/
https://ncbi.nlm.nih.gov/pubmed/22810206
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/nar/gks670
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