Polymorphic Fibrillation of the Destabilized Fourth Fasciclin-1 Domain Mutant A546T of the Transforming Growth Factor-β-induced Protein (TGFBIp) Occurs through Multiple Pathways with Different Oligomeric Intermediates

Lignende værker

• Human Phenotypically Distinct TGFBI Corneal Dystrophies Are Linked to the Stability of the Fourth FAS1 Domain of TGFBIp
  af: Runager, Kasper, et al.
  Udgivet: (2011)
• The fibril core of transforming growth factor beta-induced protein (TGFBIp) facilitates aggregation of corneal TGFBIp
  af: Sørensen, Charlotte S., et al.
  Udgivet: (2015)
• Early Events in the Amyloid Formation of the A546T Mutant of Transforming Growth Factor Beta Induced Protein (TGFBIp) in Corneal Dystrophies Compared to the Non-Fibrillating R555W and R555Q Mutants
  af: Koldsø, Heidi, et al.
  Udgivet: (2015)
• Focus on molecules: Transforming growth factor beta induced protein (TGFBIp)
  af: Runager, Kasper, et al.
  Udgivet: (2007)
• Purification, crystallization and preliminary X-ray diffraction of wild-type and mutant recombinant human transforming growth factor β-induced protein (TGFBIp)
  af: Runager, Kasper, et al.
  Udgivet: (2009)