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FAM83A confers EGFR-TKI resistance in breast cancer cells and in mice
Breast cancers commonly become resistant to EGFR–tyrosine kinase inhibitors (EGFR-TKIs); however, the mechanisms of this resistance remain largely unknown. We hypothesized that resistance may originate, at least in part, from molecular alterations that activate signaling downstream of EGFR. Using a...
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| Main Authors: | , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Clinical Investigation
2012
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3428077/ https://ncbi.nlm.nih.gov/pubmed/22886303 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI60498 |
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