Mitochondrial genome instability resulting from SUV3 haploinsufficiency leads to tumorigenesis and shortened lifespan

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• Helicase SUV3, Polynucleotide Phosphorylase, and Mitochondrial Polyadenylation Polymerase Form a Transient Complex to Modulate Mitochondrial mRNA Polyadenylated Tail Lengths in Response to Energetic Changes
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• Inactivation of CtIP Leads to Early Embryonic Lethality Mediated by G(1) Restraint and to Tumorigenesis by Haploid Insufficiency
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• Haploinsufficiency of SGO1 results in deregulated centrosome dynamics, enhanced chromosomal instability and colon tumorigenesis
  Bằng: Yamada, Hiroshi Y, et al.
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