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The T cell response to interleukin 10 alters cellular dynamics and paradoxically promotes CNS autoimmunity
IL10 is a critical anti-inflammatory cytokine, deficiency of which leads to spontaneous autoimmunity. Yet therapeutically administered or ectopically expressed IL10 may either suppress or promote disease. Distinct lineage-specific activities may explain IL10’s contradictory effects. To dissect the T...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2012
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3392541/ https://ncbi.nlm.nih.gov/pubmed/22711892 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1200607 |
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