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The receptor Ly108 functions as a SAP adaptor dependent on-off switch for T cell help to B cells and NKT cell development

Humans and mice deficient in the adaptor protein SAP (Sh2d1a) have a major defect in humoral immunity, due to lack of T cell help for B cells. The role of SAP in this process is incompletely understood. We found that deletion of receptor Ly108 (Slamf6) in CD4(+) T cells reversed the Sh2d1a(−/−) phen...

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Detalhes bibliográficos
Main Authors: Kageyama, Robin, Cannons, Jennifer L., Zhao, Fang, Yusuf, Isharat, Lao, Christopher, Locci, Michela, Schwartzberg, Pamela L., Crotty, Shane
Formato: Artigo
Idioma:Inglês
Publicado em: 2012
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3389310/
https://ncbi.nlm.nih.gov/pubmed/22683125
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.immuni.2012.05.016
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