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Impaired immune evasion in HIV through intracellular delays and multiple infection of cells
With its high mutation rate, HIV is capable of escape from recognition, suppression and/or killing by CD8(+) cytotoxic T lymphocytes (CTLs). The rate at which escape variants replace each other can give insights into the selective pressure imposed by single CTL clones. We investigate the effects of...
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| Main Authors: | , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
The Royal Society
2012
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3385478/ https://ncbi.nlm.nih.gov/pubmed/22492063 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1098/rspb.2012.0328 |
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