Induction of Fetal Hemoglobin In Vivo Mediated by a Synthetic γ-Globin Zinc Finger Activator

Sickle cell disease (SCD) and β-thalassemia patients are phenotypically normal if they carry compensatory hereditary persistence of fetal hemoglobin (HPFH) mutations that result in increased levels of fetal hemoglobin (HbF, γ-globin chains) in adulthood. Thus, research has focused on manipulating th...

पूर्ण विवरण

में बचाया:
ग्रंथसूची विवरण
मुख्य लेखकों: Costa, Flávia C., Fedosyuk, Halyna, Neades, Renee, de Los Rios, Johana Bravo, Barbas, Carlos F., Peterson, Kenneth R.
स्वरूप: Artigo
भाषा:Inglês
प्रकाशित: Hindawi Publishing Corporation 2012
विषय:
Research Article
ऑनलाइन पहुंच:https://ncbi.nlm.nih.gov/pmc/articles/PMC3384929/
https://ncbi.nlm.nih.gov/pubmed/22778925
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1155/2012/507894
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