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Prolongation of myocardial viability by proteasome inhibition during hypothermic organ preservation
Recently, we provided evidence for a possible role of the cardiac proteasome during ischemia, suggesting that a subset of 26S proteasomes is a cell-destructive protease, which is activated as the cellular energy supply declines. Although proteasome inhibition during cold ischemia (CI) reduced injury...
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| Main Authors: | , , , , , |
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| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
2010
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3384525/ https://ncbi.nlm.nih.gov/pubmed/20875792 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbrc.2010.09.093 |
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