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A subset of 26S proteasomes is activated at critically low ATP concentrations and contributes to myocardial injury during cold ischemia
Molecular mechanisms leading to myocardial injury during warm or cold ischemia are insufficiently understood. Although proteasomes are thought to contribute to myocardial ischemia-reperfusion injury, their roles during the ischemic period remain elusive. Because donor hearts are commonly exposed to...
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| Główni autorzy: | , , , , , , |
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| Format: | Artigo |
| Język: | Inglês |
| Wydane: |
2009
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| Hasła przedmiotowe: | |
| Dostęp online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3904645/ https://ncbi.nlm.nih.gov/pubmed/19944202 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbrc.2009.10.067 |
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