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Activation of TLR4 is necessary for trauma hemorrhagic shock-induced gut injury and PMN priming
Interactions of Toll-like receptors (TLR) with non-microbial factors plays a major role in the pathogenesis of early trauma-hemorrhagic shock (T/HS)-induced organ injury and inflammation. Thus, we tested the hypothesis that TLR4 mutant (TLR4(mut)) mice would be more resistant to T/HS-induced gut inj...
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| 主要な著者: | , , , , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2012
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3378823/ https://ncbi.nlm.nih.gov/pubmed/22575992 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1097/SHK.0b013e318257123a |
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