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Maternal loss of Ube3a produces an excitatory/inhibitory imbalance through neuron type-specific synaptic defects

Angelman syndrome (AS) is a neurodevelopmental disorder caused by loss of the maternally inherited allele of UBE3A. AS model mice, which carry a maternal Ube3a null mutation (Ube3a(m−/p+)), recapitulate major features of AS in humans, including enhanced seizure susceptibility. Excitatory neurotransm...

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Bibliografiske detaljer
Main Authors: Wallace, Michael L., Burette, Alain C., Weinberg, Richard J., Philpot, Benjamin D.
Format: Artigo
Sprog:Inglês
Udgivet: 2012
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3372864/
https://ncbi.nlm.nih.gov/pubmed/22681684
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuron.2012.03.036
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