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PRMT1 interacts with AML1-ETO to promote its transcriptional activation and progenitor cell proliferative potential
Fusion protein AML1-ETO, resulting from t(8;21) translocation, is highly related to leukemia development. It has been reported that full-length AML1-ETO blocks AML1 function and requires additional mutagenic events to promote leukemia. We have previously shown that the expression of AE9a, a splice i...
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| Autors principals: | , , , , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Society of Hematology
2012
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3367897/ https://ncbi.nlm.nih.gov/pubmed/22498736 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2011-04-347476 |
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