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Prion infection promotes extensive accumulation of α-synuclein in aged human α-synuclein transgenic mice
In neurodegenerative disorders of the aging population, misfolded proteins, such as PrP(Sc), α-synuclein, amyloid β protein and tau, can interact resulting in enhanced aggregation, cross seeding and accelerated disease progression. Previous reports have shown that in Creutzfeldt-Jakob disease and sc...
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| Autores principales: | , , , , , , |
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| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
Landes Bioscience
2012
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3366356/ https://ncbi.nlm.nih.gov/pubmed/22460692 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/pri.19806 |
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