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Improvements in a Mouse Model of Alzheimer’s Disease Through SOD2 Overexpression are Due to Functional and Not Structural Alterations
Oxidative stress and mitochondrial dysfunction have been implicated in the pathogenesis of Alzheimer’s disease. We and others have shown that over expression of the mitochondrial antioxidant superoxide dismutase 2 (SOD-2) can improve many of the pathologies in the Tg2576 mouse model of Alzheimer’s d...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2012
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3359050/ https://ncbi.nlm.nih.gov/pubmed/22639527 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4137/MRI.S9352 |
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