SIX1 induces lymphangiogenesis and metastasis via upregulation of VEGF-C in mouse models of breast cancer

Lignende værker

• The Six1 homeoprotein induces human mammary carcinoma cells to undergo epithelial-mesenchymal transition and metastasis in mice through increasing TGF-β signaling
  af: Micalizzi, Douglas S., et al.
  Udgivet: (2009)
• OxLDL promotes lymphangiogenesis and lymphatic metastasis in gastric cancer by upregulating VEGF-C expression and secretion
  af: Ma, Caiqi, et al.
  Udgivet: (2018)
• Expression of Six1 in luminal breast cancers predicts poor prognosis and promotes increases in tumor initiating cells by activation of extracellular signal-regulated kinase and transforming growth factor-beta signaling pathways
  af: Iwanaga, Ritsuko, et al.
  Udgivet: (2012)
• Vascular endothelial growth factor C promotes breast cancer progression via a novel antioxidant mechanism that involves regulation of superoxide dismutase 3
  af: Wang, Chu-An, et al.
  Udgivet: (2014)
• VEGF-C is positively associated with lymphangiogenesis and lymphatic metastasis in rectal cancer
  af: Pan, Zhubin, et al.
  Udgivet: (2018)