A tale of two ends

por Towers, Christina G., Ford, Heide L.
Publicado en Cell Cycle (2016)

Clonal cooperativity in heterogenous cancers

por Zhou, Hengbo, Neelakantan, Deepika, Ford, Heide L.
Publicado en Semin Cell Dev Biol (2016)

Epithelial-Mesenchymal Transition in Cancer: Parallels Between Normal Development and Tumor Progression

por Micalizzi, Douglas S., Farabaugh, Susan M., Ford, Heide L.
Publicado 2010

Intratumoral heterogeneity: Clonal cooperation in epithelial-to-mesenchymal transition and metastasis

por Neelakantan, Deepika, Drasin, David J, Ford, Heide L
Publicado en Cell Adh Migr (2014)

Cellular Plasticity in Breast Cancer Progression and Therapy

por Kong, Deguang, Hughes, Connor J., Ford, Heide L.
Publicado en Front Mol Biosci (2020)

Breast cancer epithelial-to-mesenchymal transition: examining the functional consequences of plasticity

por Drasin, David J, Robin, Tyler P, Ford, Heide L
Publicado 2011

Eya2 Is Required to Mediate the Pro-Metastatic Functions of Six1 Via the Induction of TGF-β Signaling, Epithelial-Mesenchymal Transition, and Cancer Stem Cell Properties

por Farabaugh, Susan M., Micalizzi, Douglas S., Jedlicka, Paul, Zhao, Rui, Ford, Heide L.
Publicado 2011

Vascular endothelial growth factor C promotes breast cancer progression via a novel antioxidant mechanism that involves regulation of superoxide dismutase 3

por Wang, Chu-An, Harrell, J Chuck, Iwanaga, Ritsuko, Jedlicka, Paul, Ford, Heide L
Publicado en Breast Cancer Res (2014)

The Eya Phosphatase: its unique role in cancer

por Zhou, Hengbo, Zhang, Lingdi, Vartuli, Rebecca L., Ford, Heide L., Zhao, Rui
Publicado en Int J Biochem Cell Biol (2017)

Biochemical and Functional Characterization of Six SIX1 Branchio-oto-renal Syndrome Mutations

por Patrick, Aaron N., Schiemann, Barbara J., Yang, Kui, Zhao, Rui, Ford, Heide L.
Publicado 2009

Homeoprotein Six1 Increases TGF–β Type I Receptor and Converts TGF-β Signaling from Suppressive to Supportive for Tumor Growth

por Micalizzi, Douglas S., Wang, Chu-An, Farabaugh, Susan M., Schiemann, William P., Ford, Heide L.
Publicado 2010

Distinct TRAIL resistance mechanisms can be overcome by proteasome inhibition but not generally by synergizing agents

por Menke, Christina, Bin, Lianghua, Thorburn, Jacqueline, Behbakht, Kian, Ford, Heide L., Thorburn, Andrew
Publicado 2011

MicroRNA-30a regulates zebrafish myogenesis through targeting the transcription factor Six1

por O'Brien, Jenean H., Hernandez-Lagunas, Laura, Artinger, Kristin Bruk, Ford, Heide L.
Publicado 2014

The SIX1-EYA transcriptional complex as a therapeutic target in cancer

por Blevins, Melanie A, Towers, Christina Garlington, Patrick, Aaron N., Zhao, Rui, Ford, Heide L.
Publicado en Expert Opin Ther Targets (2015)

Abrogation of the G(2) cell cycle checkpoint associated with overexpression of HSIX1: A possible mechanism of breast carcinogenesis

por Ford, Heide L., Kabingu, Edith N., Bump, Edward A., Mutter, George L., Pardee, Arthur B.
Publicado 1998

TRAIL Receptor Signaling Regulation of Chemosensitivity In Vivo but Not In Vitro

por Menke, Christina, Goncharov, Tatiana, Qamar, Lubna, Korch, Christopher, Ford, Heide L., Behbakht, Kian, Thorburn, Andrew
Publicado 2011

EWS/FLI1 regulates EYA3 in Ewing's sarcoma via modulation of microRNA-708, resulting in increased cell survival and chemoresistance

por Robin, Tyler P, Smith, Anna, McKinsey, Erin, Reaves, Lisa, Jedlicka, Paul, Ford, Heide L.
Publicado 2012

On the TRAIL to successful cancer therapy? Predicting and counteracting resistance against TRAIL-based therapeutics

por Dimberg, Lina Y., Anderson, Charles K., Camidge, Ross, Behbakht, Kian, Thorburn, Andrew, Ford, Heide L.
Publicado en Oncogene (2012)

Structure-Function Analyses of the Human SIX1–EYA2 Complex Reveal Insights into Metastasis and BOR Syndrome

por Patrick, Aaron N, Cabrera, Joshua H, Smith, Anna L, Chen, Xiaojiang S, Ford, Heide L, Zhao, Rui
Publicado 2013

Negative regulation of endothelin signaling by SIX1 is required for proper maxillary development

por Tavares, Andre L. P., Cox, Timothy C., Maxson, Robert M., Ford, Heide L., Clouthier, David E.
Publicado en Development (2017)