ENVIRONMENTAL NEUROTOXIC PESTICIDE DIELDRIN ACTIVATES A NON RECEPTOR TYROSINE KINASE TO PROMOTE PKCδ-MEDIATED DOPAMINERGIC APOPTOSIS IN A DOPAMINERGIC NEURONAL CELL MODEL

Lignende værker

• Protein kinase D1 (PKD1) activation mediates a compensatory protective response during early stages of oxidative stress-induced neuronal degeneration
  af: Anantharam Vellareddy, et al.
  Udgivet: (2011-06-01)
• Protein kinase D1 (PKD1) activation mediates a compensatory protective response during early stages of oxidative stress-induced neuronal degeneration
  af: Asaithambi, Arunkumar, et al.
  Udgivet: (2011)
• Methamphetamine-induced neurotoxicity linked to UPS dysfunction and autophagy related changes that can be modulated by PKCδ in dopaminergic neuronal cells
  af: Lin, Mengshien, et al.
  Udgivet: (2012)
• Fyn Kinase-Mediated PKCδ Y311 Phosphorylation Induces Dopaminergic Degeneration in Cell Culture and Animal Models: Implications for the Identification of a New Pharmacological Target for Parkinson’s Disease
  af: Saminathan, Hariharan, et al.
  Udgivet: (2021)
• Dopaminergic Neurotoxicant 6-OHDA Induces Oxidative Damage through Proteolytic Activation of PKCδ in Cell Culture and Animal Models of Parkinson’s Disease
  af: Latchoumycandane, Calivarathan, et al.
  Udgivet: (2011)