Insulin and AMPK regulate FA translocase/CD36 plasma membrane recruitment in cardiomyocytes via Rab GAP AS160 and Rab8a Rab GTPase

The FA translocase cluster of differentiation 36 (CD36) facilitates FA uptake by the myocardium, and its surface recruitment in cardiomyocytes is induced by insulin, AMP-dependent protein kinase (AMPK), or contraction. Dysfunction of CD36 trafficking contributes to disordered cardiac FA utilization...

Full beskrivning

Sparad:
Bibliografiska uppgifter
Huvudupphovsmän: Samovski, Dmitri, Su, Xiong, Xu, Yingcheng, Abumrad, Nada A., Stahl, Philip D.
Materialtyp: Artigo
Språk:Inglês
Publicerad: The American Society for Biochemistry and Molecular Biology 2012
Ämnen:
Research Articles
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC3307647/
https://ncbi.nlm.nih.gov/pubmed/22315395
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1194/jlr.M023424
Taggar: Lägg till en tagg
Inga taggar, Lägg till första taggen!

Liknande verk