Insulin and AMPK regulate FA translocase/CD36 plasma membrane recruitment in cardiomyocytes via Rab GAP AS160 and Rab8a Rab GTPase

The FA translocase cluster of differentiation 36 (CD36) facilitates FA uptake by the myocardium, and its surface recruitment in cardiomyocytes is induced by insulin, AMP-dependent protein kinase (AMPK), or contraction. Dysfunction of CD36 trafficking contributes to disordered cardiac FA utilization...

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Detalhes bibliográficos
Main Authors: Samovski, Dmitri, Su, Xiong, Xu, Yingcheng, Abumrad, Nada A., Stahl, Philip D.
Formato: Artigo
Idioma:Inglês
Publicado em: The American Society for Biochemistry and Molecular Biology 2012
Assuntos:
Research Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3307647/
https://ncbi.nlm.nih.gov/pubmed/22315395
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1194/jlr.M023424
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