Insulin and AMPK regulate FA translocase/CD36 plasma membrane recruitment in cardiomyocytes via Rab GAP AS160 and Rab8a Rab GTPase

The FA translocase cluster of differentiation 36 (CD36) facilitates FA uptake by the myocardium, and its surface recruitment in cardiomyocytes is induced by insulin, AMP-dependent protein kinase (AMPK), or contraction. Dysfunction of CD36 trafficking contributes to disordered cardiac FA utilization...

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Библиографические подробности
Главные авторы: Samovski, Dmitri, Su, Xiong, Xu, Yingcheng, Abumrad, Nada A., Stahl, Philip D.
Формат: Artigo
Язык:Inglês
Опубликовано: The American Society for Biochemistry and Molecular Biology 2012
Предметы:
Research Articles
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC3307647/
https://ncbi.nlm.nih.gov/pubmed/22315395
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1194/jlr.M023424
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