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Akt-dependent Skp2 mRNA translation is required for exiting contact inhibition, oncogenesis, and adipogenesis

The requirement of Akt for cell proliferation and oncogenesis is mammalian target of rapamycin complex 1 (mTORC1) dependent. SV40 large T expression in Akt-deficient cells restores cell proliferation rate, but is insufficient for exiting contact inhibition and oncogene-induced anchorage-independent...

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Detalhes bibliográficos
Main Authors: Nogueira, Veronique, Sundararajan, Deepa, Kwan, Jennifer M, Peng, Xiao-ding, Sarvepalli, Neha, Sonenberg, Nahum, Hay, Nissim
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group 2012
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3297989/
https://ncbi.nlm.nih.gov/pubmed/22307088
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/emboj.2011.478
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