Angiotensin type I receptor blockade in conjunction with enhanced Akt activation restores coronary collateral growth in the metabolic syndrome

We have previously demonstrated that Akt was required for repetitive ischemia (RI)-induced coronary collateral growth (CCG) in healthy rats but was not activated by RI in the metabolic syndrome (JCR:LA-cp rats) where CCG was impaired. Here we hypothesized that failure of angiotensin type I receptor...

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Main Authors: Jadhav, Rashmi, Dodd, Tracy, Smith, Erika, Bailey, Erin, DeLucia, Angelo L., Russell, James C., Madison, Rowan, Potter, Barry, Walsh, Kenneth, Jo, Hanjoong, Rocic, Petra
Format: Artigo
Sprog:Inglês
Udgivet: American Physiological Society 2011
Fag:
Integrative Cardiovascular Physiology and Pathophysiology
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3283040/
https://ncbi.nlm.nih.gov/pubmed/21335466
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00282.2010
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