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Redox-sensitive Akt and Src regulate coronary collateral growth in metabolic syndrome

We have recently shown that the inability of repetitive ischemia (RI) to activate p38 MAPK (p38) and Akt in metabolic syndrome [JCR:LA-cp (JCR)] rats was associated with impaired coronary collateral growth (CCG). Furthermore, Akt and p38 activation correlated with optimal O(2)(−)· levels and were al...

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Autors principals: Reed, Ryan, Potter, Barry, Smith, Erika, Jadhav, Rashmi, Villalta, Patricia, Jo, Hanjoong, Rocic, Petra
Format: Artigo
Idioma:Inglês
Publicat: American Physiological Society 2009
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC2716105/
https://ncbi.nlm.nih.gov/pubmed/19376806
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00920.2008
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