Redox-sensitive Akt and Src regulate coronary collateral growth in metabolic syndrome

Por Reed, Ryan, Potter, Barry, Smith, Erika, Jadhav, Rashmi, Villalta, Patricia, Jo, Hanjoong, Rocic, Petra
Publicado em 2009

MMPs 2 and 9 are essential for coronary collateral growth and are prominently regulated by p38 MAPK

Por Dodd, Tracy, Jadhav, Rashmi, Wiggins, Luke, Stewart, James, Smith, Erika, Russell, James C., Rocic, Petra
Publicado em 2011

Slingshot isoform-specific regulation of cofilin-mediated vascular smooth muscle cell migration and neointima formation

Por Torres, Rebecca A., Drake, Douglas A., Solodushko, Viktoriya, Jadhav, Rashmi, Smith, Erika, Rocic, Petra, Weber, David S.
Publicado em 2011

miR-21-mediated decreased neutrophil apoptosis is a determinant of impaired coronary collateral growth in metabolic syndrome

Por Hutcheson, Rebecca, Terry, Russell, Hutcheson, Brenda, Jadhav, Rashmi, Chaplin, Jennifer, Smith, Erika, Barrington, Robert, Proctor, Spencer D., Rocic, Petra
Publicado no Am J Physiol Heart Circ Physiol (2015)

miR-21 normalizes vascular smooth muscle proliferation and improves coronary collateral growth in metabolic syndrome

Por Hutcheson, Rebecca, Chaplin, Jennifer, Hutcheson, Brenda, Borthwick, Faye, Proctor, Spencer, Gebb, Sarah, Jadhav, Rashmi, Smith, Erika, Russell, James C., Rocic, Petra
Publicado em 2014

Angiotensin type I receptor blockade in conjunction with enhanced Akt activation restores coronary collateral growth in the metabolic syndrome

Por Jadhav, Rashmi, Dodd, Tracy, Smith, Erika, Bailey, Erin, DeLucia, Angelo L., Russell, James C., Madison, Rowan, Potter, Barry, Walsh, Kenneth, Jo, Hanjoong, Rocic, Petra
Publicado em 2011