Arrhythmogenic consequences of myofibroblast–myocyte coupling

AIMS: Fibrosis is known to promote cardiac arrhythmias by disrupting myocardial structure. Given recent evidence that myofibroblasts form gap junctions with myocytes at least in co-cultures, we investigated whether myofibroblast–myocyte coupling can promote arrhythmia triggers, such as early afterde...

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Detaylı Bibliyografya
Asıl Yazarlar: Nguyen, Thao P., Xie, Yuanfang, Garfinkel, Alan, Qu, Zhilin, Weiss, James N.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Oxford University Press 2012
Konular:
Original Articles
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3258652/
https://ncbi.nlm.nih.gov/pubmed/22049532
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/cvr/cvr292
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