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Arrhythmogenic consequences of myofibroblast–myocyte coupling
AIMS: Fibrosis is known to promote cardiac arrhythmias by disrupting myocardial structure. Given recent evidence that myofibroblasts form gap junctions with myocytes at least in co-cultures, we investigated whether myofibroblast–myocyte coupling can promote arrhythmia triggers, such as early afterde...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Oxford University Press
2012
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3258652/ https://ncbi.nlm.nih.gov/pubmed/22049532 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/cvr/cvr292 |
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