Creatine kinase–mediated improvement of function in failing mouse hearts provides causal evidence the failing heart is energy starved

ATP is required for normal cardiac contractile function, and it has long been hypothesized that reduced energy delivery contributes to the contractile dysfunction of heart failure (HF). Despite experimental and clinical HF data showing reduced metabolism through cardiac creatine kinase (CK), the maj...

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Autors principals: Gupta, Ashish, Akki, Ashwin, Wang, Yibin, Leppo, Michelle K., Chacko, V.P., Foster, D. Brian, Caceres, Viviane, Shi, Sa, Kirk, Jonathan A., Su, Jason, Lai, Shenghan, Paolocci, Nazareno, Steenbergen, Charles, Gerstenblith, Gary, Weiss, Robert G.
Format: Artigo
Idioma:Inglês
Publicat: American Society for Clinical Investigation 2011
Matèries:
Research Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3248286/
https://ncbi.nlm.nih.gov/pubmed/22201686
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI57426
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