Creatine kinase–mediated improvement of function in failing mouse hearts provides causal evidence the failing heart is energy starved

ATP is required for normal cardiac contractile function, and it has long been hypothesized that reduced energy delivery contributes to the contractile dysfunction of heart failure (HF). Despite experimental and clinical HF data showing reduced metabolism through cardiac creatine kinase (CK), the maj...

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書誌詳細
主要な著者: Gupta, Ashish, Akki, Ashwin, Wang, Yibin, Leppo, Michelle K., Chacko, V.P., Foster, D. Brian, Caceres, Viviane, Shi, Sa, Kirk, Jonathan A., Su, Jason, Lai, Shenghan, Paolocci, Nazareno, Steenbergen, Charles, Gerstenblith, Gary, Weiss, Robert G.
フォーマット: Artigo
言語:Inglês
出版事項: American Society for Clinical Investigation 2011
主題:
Research Article
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC3248286/
https://ncbi.nlm.nih.gov/pubmed/22201686
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI57426
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