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Creatine kinase–mediated improvement of function in failing mouse hearts provides causal evidence the failing heart is energy starved
ATP is required for normal cardiac contractile function, and it has long been hypothesized that reduced energy delivery contributes to the contractile dysfunction of heart failure (HF). Despite experimental and clinical HF data showing reduced metabolism through cardiac creatine kinase (CK), the maj...
Gorde:
| Egile Nagusiak: | , , , , , , , , , , , , , , |
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| Formatua: | Artigo |
| Hizkuntza: | Inglês |
| Argitaratua: |
American Society for Clinical Investigation
2011
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| Gaiak: | |
| Sarrera elektronikoa: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3248286/ https://ncbi.nlm.nih.gov/pubmed/22201686 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI57426 |
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