The carboxy-terminal fragment of inhibitor-2 of protein phosphatase-2A induces Alzheimer disease pathology and cognitive impairment

Development of rational therapeutic treatments of Alzheimer disease (AD) requires the elucidation of the etiopathogenic mechanisms of neurofibrillary degeneration and β-amyloidosis, the two hallmarks of this disease. Here we show, employing an adeno-associated virus serotype 1 (AAV1)-induced express...

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Autors principals: Wang, Xiaochuan, Blanchard, Julie, Kohlbrenner, Erik, Clement, Nathalie, Linden, R. Michael, Radu, Aurelian, Grundke-Iqbal, Inge, Iqbal, Khalid
Format: Artigo
Idioma:Inglês
Publicat: Federation of American Societies for Experimental Biology 2010
Matèries:
Research Communications
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3229424/
https://ncbi.nlm.nih.gov/pubmed/20651003
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.10-158477
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