A20-binding inhibitor of NF-κB (ABIN1) controls Toll-like receptor-mediated CCAAT/enhancer-binding protein β activation and protects from inflammatory disease

Toll-like receptors (TLRs) are expressed on innate immune cells and trigger inflammation upon detection of pathogens and host tissue injury. TLR-mediated proinflammatory-signaling pathways are counteracted by partially characterized anti-inflammatory mechanisms that prevent exaggerated inflammation...

תיאור מלא

שמור ב:
מידע ביבליוגרפי
Main Authors: Zhou, Jingran, Wu, Ruiqiong, High, Anthony A., Slaughter, Clive A., Finkelstein, David, Rehg, Jerold E., Redecke, Vanessa, Häcker, Hans
פורמט: Artigo
שפה:Inglês
יצא לאור: National Academy of Sciences 2011
נושאים:
PNAS Plus
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC3207663/
https://ncbi.nlm.nih.gov/pubmed/22011580
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1106232108
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