Neuroprotection against Traumatic Brain Injury by a Peptide Derived from the Collapsin Response Mediator Protein 2 (CRMP2)

Neurological disabilities following traumatic brain injury (TBI) may be due to excitotoxic neuronal loss. The excitotoxic loss of neurons following TBI occurs largely due to hyperactivation of N-methyl-d-aspartate receptors (NMDARs), leading to toxic levels of intracellular Ca(2+). The axon guidance...

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Bibliographic Details
Main Authors: Brittain, Joel M., Chen, Liang, Wilson, Sarah M., Brustovetsky, Tatiana, Gao, Xiang, Ashpole, Nicole M., Molosh, Andrei I., You, Haitao, Hudmon, Andy, Shekhar, Anantha, White, Fletcher A., Zamponi, Gerald W., Brustovetsky, Nickolay, Chen, Jinhui, Khanna, Rajesh
Format: Artigo
Language:Inglês
Published: American Society for Biochemistry and Molecular Biology 2011
Subjects:
Neurobiology
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC3199520/
https://ncbi.nlm.nih.gov/pubmed/21832084
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M111.255455
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