Neuroprotection against Traumatic Brain Injury by a Peptide Derived from the Collapsin Response Mediator Protein 2 (CRMP2)

Neurological disabilities following traumatic brain injury (TBI) may be due to excitotoxic neuronal loss. The excitotoxic loss of neurons following TBI occurs largely due to hyperactivation of N-methyl-d-aspartate receptors (NMDARs), leading to toxic levels of intracellular Ca(2+). The axon guidance...

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Detalhes bibliográficos
Main Authors: Brittain, Joel M., Chen, Liang, Wilson, Sarah M., Brustovetsky, Tatiana, Gao, Xiang, Ashpole, Nicole M., Molosh, Andrei I., You, Haitao, Hudmon, Andy, Shekhar, Anantha, White, Fletcher A., Zamponi, Gerald W., Brustovetsky, Nickolay, Chen, Jinhui, Khanna, Rajesh
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Biochemistry and Molecular Biology 2011
Assuntos:
Neurobiology
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3199520/
https://ncbi.nlm.nih.gov/pubmed/21832084
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M111.255455
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