Neuroprotection against Traumatic Brain Injury by a Peptide Derived from the Collapsin Response Mediator Protein 2 (CRMP2)

Neurological disabilities following traumatic brain injury (TBI) may be due to excitotoxic neuronal loss. The excitotoxic loss of neurons following TBI occurs largely due to hyperactivation of N-methyl-d-aspartate receptors (NMDARs), leading to toxic levels of intracellular Ca(2+). The axon guidance...

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Main Authors: Brittain, Joel M., Chen, Liang, Wilson, Sarah M., Brustovetsky, Tatiana, Gao, Xiang, Ashpole, Nicole M., Molosh, Andrei I., You, Haitao, Hudmon, Andy, Shekhar, Anantha, White, Fletcher A., Zamponi, Gerald W., Brustovetsky, Nickolay, Chen, Jinhui, Khanna, Rajesh
פורמט: Artigo
שפה:Inglês
יצא לאור: American Society for Biochemistry and Molecular Biology 2011
נושאים:
Neurobiology
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC3199520/
https://ncbi.nlm.nih.gov/pubmed/21832084
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M111.255455
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