Herpes simplex virus encephalitis in a patient with complete TLR3 deficiency: TLR3 is otherwise redundant in protective immunity

Autosomal dominant TLR3 deficiency has been identified as a genetic etiology of childhood herpes simplex virus 1 (HSV-1) encephalitis (HSE). This defect is partial, as it results in impaired, but not abolished induction of IFN-β and -λ in fibroblasts in response to TLR3 stimulation. The apparently n...

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Main Authors: Guo, Yiqi, Audry, Magali, Ciancanelli, Michael, Alsina, Laia, Azevedo, Joana, Herman, Melina, Anguiano, Esperanza, Sancho-Shimizu, Vanessa, Lorenzo, Lazaro, Pauwels, Elodie, Philippe, Paul Bastard, Pérez de Diego, Rebeca, Cardon, Annabelle, Vogt, Guillaume, Picard, Capucine, Andrianirina, Zafitsara Zo, Rozenberg, Flore, Lebon, Pierre, Plancoulaine, Sabine, Tardieu, Marc, Valérie Doireau, Jouanguy, Emmanuelle, Chaussabel, Damien, Geissmann, Frederic, Abel, Laurent, Casanova, Jean-Laurent, Zhang, Shen-Ying
פורמט: Artigo
שפה:Inglês
יצא לאור: The Rockefeller University Press 2011
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Article
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC3182056/
https://ncbi.nlm.nih.gov/pubmed/21911422
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20101568
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