Herpes simplex virus encephalitis in a patient with complete TLR3 deficiency: TLR3 is otherwise redundant in protective immunity

Autosomal dominant TLR3 deficiency has been identified as a genetic etiology of childhood herpes simplex virus 1 (HSV-1) encephalitis (HSE). This defect is partial, as it results in impaired, but not abolished induction of IFN-β and -λ in fibroblasts in response to TLR3 stimulation. The apparently n...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Guo, Yiqi, Audry, Magali, Ciancanelli, Michael, Alsina, Laia, Azevedo, Joana, Herman, Melina, Anguiano, Esperanza, Sancho-Shimizu, Vanessa, Lorenzo, Lazaro, Pauwels, Elodie, Philippe, Paul Bastard, Pérez de Diego, Rebeca, Cardon, Annabelle, Vogt, Guillaume, Picard, Capucine, Andrianirina, Zafitsara Zo, Rozenberg, Flore, Lebon, Pierre, Plancoulaine, Sabine, Tardieu, Marc, Valérie Doireau, Jouanguy, Emmanuelle, Chaussabel, Damien, Geissmann, Frederic, Abel, Laurent, Casanova, Jean-Laurent, Zhang, Shen-Ying
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: The Rockefeller University Press 2011
Aiheet:
Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3182056/
https://ncbi.nlm.nih.gov/pubmed/21911422
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20101568
Tagit: Lisää tagi
Ei tageja, Lisää ensimmäinen tagi!

Samankaltaisia teoksia