Herpes simplex virus encephalitis in a patient with complete TLR3 deficiency: TLR3 is otherwise redundant in protective immunity

Autosomal dominant TLR3 deficiency has been identified as a genetic etiology of childhood herpes simplex virus 1 (HSV-1) encephalitis (HSE). This defect is partial, as it results in impaired, but not abolished induction of IFN-β and -λ in fibroblasts in response to TLR3 stimulation. The apparently n...

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التفاصيل البيبلوغرافية
المؤلفون الرئيسيون: Guo, Yiqi, Audry, Magali, Ciancanelli, Michael, Alsina, Laia, Azevedo, Joana, Herman, Melina, Anguiano, Esperanza, Sancho-Shimizu, Vanessa, Lorenzo, Lazaro, Pauwels, Elodie, Philippe, Paul Bastard, Pérez de Diego, Rebeca, Cardon, Annabelle, Vogt, Guillaume, Picard, Capucine, Andrianirina, Zafitsara Zo, Rozenberg, Flore, Lebon, Pierre, Plancoulaine, Sabine, Tardieu, Marc, Valérie Doireau, Jouanguy, Emmanuelle, Chaussabel, Damien, Geissmann, Frederic, Abel, Laurent, Casanova, Jean-Laurent, Zhang, Shen-Ying
التنسيق: Artigo
اللغة:Inglês
منشور في: The Rockefeller University Press 2011
الموضوعات:
Article
الوصول للمادة أونلاين:https://ncbi.nlm.nih.gov/pmc/articles/PMC3182056/
https://ncbi.nlm.nih.gov/pubmed/21911422
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20101568
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