Nalaganje...
Oppositely imprinted genes p57(Kip2) and Igf2 interact in a mouse model for Beckwith–Wiedemann syndrome
Beckwith–Wiedemann syndrome (BWS) is a clinically variable disorder characterized by somatic overgrowth, macroglossia, abdominal wall defects, visceromegaly, and an increased susceptibility to childhood tumors. The disease has been linked to a large cluster of imprinted genes at human chromosome 11p...
Shranjeno v:
| izdano v: | Genes Dev |
|---|---|
| Main Authors: | , , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
Cold Spring Harbor Laboratory Press
1999
|
| Teme: | |
| Online dostop: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC317182/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10601037/ https://ncbi.nlm.nih.govhttps://doi.org/10.1101/gad.13.23.3115 |
| Oznake: |
Označite
Brez oznak, prvi označite!
|