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Oppositely imprinted genes p57(Kip2) and Igf2 interact in a mouse model for Beckwith–Wiedemann syndrome

Beckwith–Wiedemann syndrome (BWS) is a clinically variable disorder characterized by somatic overgrowth, macroglossia, abdominal wall defects, visceromegaly, and an increased susceptibility to childhood tumors. The disease has been linked to a large cluster of imprinted genes at human chromosome 11p...

Disgrifiad llawn

Wedi'i Gadw mewn:
Manylion Llyfryddiaeth
Prif Awduron: Caspary, Tamara, Cleary, Michele A., Perlman, Elizabeth J., Zhang, Pumin, Elledge, Stephen J., Tilghman, Shirley M.
Fformat: Artigo
Iaith:Inglês
Cyhoeddwyd: Cold Spring Harbor Laboratory Press 1999
Pynciau:
Mynediad Ar-lein:https://ncbi.nlm.nih.gov/pmc/articles/PMC317182/
https://ncbi.nlm.nih.gov/pubmed/10601037
Tagiau: Ychwanegu Tag
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