Oppositely imprinted genes p57(Kip2) and Igf2 interact in a mouse model for Beckwith–Wiedemann syndrome

Beckwith–Wiedemann syndrome (BWS) is a clinically variable disorder characterized by somatic overgrowth, macroglossia, abdominal wall defects, visceromegaly, and an increased susceptibility to childhood tumors. The disease has been linked to a large cluster of imprinted genes at human chromosome 11p...

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Dades bibliogràfiques

Publicat a:	Genes Dev
Autors principals:	Caspary, Tamara, Cleary, Michele A., Perlman, Elizabeth J., Zhang, Pumin, Elledge, Stephen J., Tilghman, Shirley M.
Format:	Artigo
Idioma:	Inglês
Publicat:	Cold Spring Harbor Laboratory Press 1999
Matèries:	Research Paper
Accés en línia:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC317182/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10601037/ https://ncbi.nlm.nih.govhttps://doi.org/10.1101/gad.13.23.3115
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