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β-Cell-specific inactivation of the mouse Ipf1/Pdx1 gene results in loss of the β-cell phenotype and maturity onset diabetes

To study the late β-cell-specific function of the homeodomain protein IPF1/PDX1 we have generated mice in which the Ipf1/Pdx1 gene has been disrupted specifically in β cells. These mice develop diabetes with age, and we show that IPF1/PDX1 is required for maintaining the β cell identity by positivel...

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Detalhes bibliográficos
Main Authors: Ahlgren, Ulf, Jonsson, Jörgen, Jonsson, Lena, Simu, Karin, Edlund, Helena
Formato: Artigo
Idioma:Inglês
Publicado em: Cold Spring Harbor Laboratory Press 1998
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC316911/
https://ncbi.nlm.nih.gov/pubmed/9637677
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